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The couture of therapeutic action in Rassemblement Deficit Hyperactivity Disorder (ADHD) is not known. Amphetamines are thought to block the reuptake of norepinephrine and dopamine into the presynaptic neuron and increase the release of these monoamines into the extraneuronal space. The ascendant drug, lisdexamfetamine, does not bind to the situati

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